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Respiratory Muscle Strength and Control of Ventilation: Discussion (9)

Posted in  Tuesday, March 19th 2013
Topics: Pulmonary function     Tags: dystrophy, lung volumes, neuromuscular disease, respiratory muscles

Another mechanism could be increased stimulation or irritation of chest wall and airway receptors generating an increase in Po.i. In this study, there was no statistically significant correlation between PaC02 and Po.i. Finally, it has been shown that respiratory muscle fatigue is less likely to occur with submaximal than with maximal Ve. If this phenomenon […]

Respiratory Muscle Strength and Control of Ventilation: Discussion (8)

Posted in  Monday, March 18th 2013
Topics: Pulmonary function     Tags: dystrophy, lung volumes, neuromuscular disease, respiratory muscles

Transdiaphragmatic pressure swings (APdi) during quiet breathing in patients with myotonic dystrophy are nearly three times that of normal seated subjects and are probably related to greater impedance to diaphragmatic motion caused either by chest wall stiffness or increase of tone in intercostal and abdominal wall muscles. Patients with muscular dystrophy and other paralytic neuromuscular […]

Respiratory Muscle Strength and Control of Ventilation: Discussion (7)

Posted in  Sunday, March 17th 2013
Topics: Pulmonary function     Tags: dystrophy, lung volumes, neuromuscular disease, respiratory muscles

The increased Po.i relative to Ve measurements in the group with neuromuscular disease demonstrates that these two measurements are not comparable under these circumstances. In a study of eight normal volunteers undergoing C02 rebreathing during progressive curarization, Holle and coworkers also found that despite increasing muscle weakness and decreasing Ve, Po.i did not decrease, and […]

Respiratory Muscle Strength and Control of Ventilation: Discussion (6)

Posted in  Saturday, March 16th 2013
Topics: Pulmonary function     Tags: dystrophy, lung volumes, neuromuscular disease, respiratory muscles

In fact, in the patients with neuromuscular disease, Po.i ranged between 0.72 and 3.59 cm H20, while Pimax at FRC averaged 43 percent of the control mean. Similar augmentation of the central drive to breathe has been documented in other chronic chest wall disorders without C02 retention, such as obesity. The breathing pattern of our […]

Respiratory Muscle Strength and Control of Ventilation: Discussion (5)

Posted in  Friday, March 15th 2013
Topics: Pulmonary function     Tags: dystrophy, lung volumes, neuromuscular disease, respiratory muscles

In addition, microatelectasis, by reducing the volume of gas remaining in the lungs at the end of a maximal expiration, may also tend to counter the increase in RV that would otherwise occur. In our study, the patients with neuromuscular disease demonstrated VC and ERV values that were, on the average, about one third of […]

Respiratory Muscle Strength and Control of Ventilation: Discussion (4)

Posted in  Thursday, March 14th 2013
Topics: Pulmonary function     Tags: dystrophy, lung volumes, neuromuscular disease, respiratory muscles

In contrast, Ers in the patients with neuromuscular disease averaged 0.27 cmH20/percent VC, similar to the Ers of 0.19 cm H20/percent VC in seated conscious patients with neuromuscular disease whose cases were reported by Gibson et al. These findings imply that the increase in Ers observed in our patients with neuromuscular disease is proportional to […]

Respiratory Muscle Strength and Control of Ventilation: Discussion (3)

Posted in  Wednesday, March 13th 2013
Topics: Pulmonary function     Tags: dystrophy, lung volumes, neuromuscular disease, respiratory muscles

(c) Most Pmax values are recorded at one lung volume, usually FRC, and, as such, are regarded as quasi-static measurements during which flow rates are considered to be negligible. Because of flow- and volume-related losses in patients with neuromuscular disease, Pmax measurements may fail to detect an inability to cope with the increased ventilatory demands […]

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